Ketogenic Diet for Cancer: A Naturopathic Oncologist's Full Guide
- Dr. Lena Suhaila
- Jun 12
- 16 min read
By Dr. Lena Suhaila, ND, FABNO
A ketogenic diet is very low in carbohydrate, moderate in protein, and high in healthy fat, which shifts your body from burning glucose to burning fat-derived ketones. It's directly relevant to cancer because most cancer cells depend on glucose and can't efficiently use ketones for fuel, so lowering glucose while raising ketones gives your healthy cells an energy advantage these cells can't match. This is a clinical approach with more than a century of use in medicine, and the science behind it speaks to anyone in active treatment, recovery, or prevention.
This is not a weight loss article. This is about metabolism, about how cancer cells eat, and about a dietary approach that has been used in clinical medicine for over 100 years. It builds on the foundation I laid out in Food as Metabolic Medicine.
What is the ketogenic diet?
The ketogenic diet is very low in carbohydrates, moderate in protein, and high in healthy fats. When you consistently keep carbohydrates under 30 grams of net carbs per day, your body runs out of glucose to burn and shifts to burning fat instead. Your liver produces molecules called ketone bodies in the process, and these become your primary fuel source. This metabolic state is called ketosis.
The three main ketone bodies are beta-hydroxybutyrate (BHB), acetoacetate, and acetone. BHB is the most abundant and is what you measure when you test blood ketones at home with a meter.
The macronutrient breakdown on a therapeutic ketogenic diet looks roughly like this: about 70 to 75% of calories from fat, 20 to 25% from protein, and 5% from carbohydrates. Protein is kept moderate rather than high, because excess protein can convert to glucose through a process called gluconeogenesis, which would work against therapeutic ketosis. For most patients I work with, the protein target sits between 1.0 and 1.2 grams per kilogram of actual body weight per day. That range protects lean muscle mass without pushing you out of ketosis. During active treatment, in patients at risk of cachexia, or in athletes, this number goes higher under clinical supervision. Cancer-specific guidance from organizations like ESPEN recommends 1.0 to 1.5 grams of protein per kilogram of actual body weight in cancer patients, and the ketogenic version of this guidance sits in the lower portion of that range.
Healthy cells can run on either glucose or fat-derived ketones. Most cancer cells can only run on glucose. A ketogenic diet lowers glucose and raises ketones, giving your healthy cells an energy advantage that cancer cells cannot match.
How does ketosis work in your body?
On a standard diet, every time you eat bread, pasta, rice, fruit, or anything sweet, your digestive system breaks it into glucose, your blood sugar rises, and your pancreas releases insulin to push that glucose into your cells for energy. Blood sugar goes up, insulin spikes, blood sugar comes back down. This happens multiple times every day.
When you remove carbohydrates from your diet, a cascade of events follows. Blood glucose drops to a lower, more stable level. Insulin falls significantly. Your liver begins converting both dietary fat and stored body fat into ketones, which then circulate in the bloodstream and fuel most of your cells, including your brain.
After two to four weeks of consistent low-carbohydrate eating, your body becomes what is called fat-adapted. Your cells become efficient at running on fat and ketones rather than glucose. Most people at this stage report steadier energy throughout the day, clearer thinking, and noticeably less hunger, particularly between meals.
Target ranges to know
Blood ketone levels tell you how deeply into ketosis you are. These targets are the ones we use in clinical practice. Nutritional ketosis runs from 0.5 to 3.0 mmol/L blood ketones. My therapeutic target for cancer support is 2.0 to 3.0 mmol/L, the upper end of that range. Fasting glucose, in active treatment and in recovery alike, is 70 to 85 mg/dL.
Use a blood ketone meter such as the KetoMojo. Urine ketone strips become unreliable once you are fat-adapted and stop detecting ketones even when you are firmly in ketosis. Blood testing is the only reliable method for therapeutic monitoring.
The Warburg Effect: why keto is relevant in cancer care
In the 1920s, German biochemist Otto Warburg discovered something unusual about cancer cells. Unlike healthy cells, which produce energy efficiently using oxygen, most cancer cells rely almost exclusively on glucose for fuel, fermenting it rather than burning it cleanly. This characteristic is now called the Warburg Effect, and it has been documented across dozens of tumor types. [1]
The critical detail is this: cancer cells have severely limited ability to use ketone bodies as fuel. They lack the metabolic flexibility that healthy cells have. When you lower blood glucose and raise blood ketones through a ketogenic diet, you create an environment that deprives cancer cells of their preferred fuel while your healthy cells run efficiently on the alternative source. I walk through the cell-level mechanics in more depth in How Ketogenic Diets Target Cancer Cells.
The Warburg Effect itself is a well-characterized feature of cancer cell biology and forms the metabolic rationale for the ketogenic diet in oncology. The clinical research applying that rationale through dietary intervention is still developing, and the studies cited below reflect where that evidence currently stands.
What does the clinical research show?
A 2024 systematic review and meta-analysis published in Nutrition Bulletin (PMID: 38923748) examined randomized controlled trials of the ketogenic diet in cancer patients and found meaningful reductions in blood glucose levels, with tumor-related improvements seen in several studies when the diet was combined with standard treatment. [2]
A 2025 meta-analysis published in Frontiers in Nutrition (PMID: 40756563), covering studies through June 2024, found that compared to standard diets, the ketogenic diet significantly reduced fat mass, visceral fat, insulin levels, blood glucose, fatigue, and insomnia in cancer patients. It also showed meaningful improvements in emotional functioning and social functioning scores. [3]
Animal research by Klement and colleagues, analyzing survival data from over 1,700 mice across multiple tumor types, found that the ketogenic diet alone significantly extended survival, and that combining it with radiation therapy extended survival further still. [4]
How does keto help with cancer treatment side effects?
One of the most practical reasons I recommend this approach during cancer treatment is what it does to the side effects that make treatment so hard to get through.
Side Effect | How the Ketogenic Diet Helps |
Fatigue | Ketones provide stable, consistent fuel without blood sugar crashes. The 2025 meta-analysis found statistically significant reductions in fatigue scores in cancer patients on a ketogenic diet. |
Nausea | High-fat, low-carbohydrate foods are generally better tolerated during chemotherapy. Avocado, eggs, broth, and coconut-based foods sit well on difficult days. |
Brain fog | Ketones are neuroprotective and provide an alternate fuel source that bypasses impaired glucose transport mechanisms contributing to cognitive difficulties during treatment. |
Muscle wasting | Abundant fat fuel reduces the body's need to break down muscle for energy. Lower inflammatory cytokines associated with ketogenic eating also reduce the cachexia pathway. |
Steroid-related blood sugar spikes | Corticosteroids used in cancer care cause significant glucose spikes. Carbohydrate restriction powerfully counteracts this effect and reduces the glucose burden that feeds tumor cells. |
Insomnia | Stable blood glucose overnight reduces cortisol surges that fragment sleep. The 2025 meta-analysis found significant reduction in insomnia scores among cancer patients on a ketogenic diet. |
Inflammation | BHB inhibits the NLRP3 inflammasome, one of the primary drivers of chronic inflammation underlying both tumor progression and treatment-related pain and swelling. |
Remove added sugars and refined starches from your diet today. This single step lowers insulin, reduces inflammation, and stabilizes blood sugar. Your morning fasting blood glucose number is the single most informative daily measurement you can take.
How do you start a ketogenic diet?
What follows is a starting framework, not a prescription. The ketone and glucose targets in this article are not appropriate for every patient, particularly during active treatment, and how aggressively you pursue them is a clinical decision that should be made with a clinician who knows your specific situation, treatment regimen, medications, and current nutritional status. What follows works best as a conversation starter with that clinician. People who are underweight, who have Type 1 diabetes, significant kidney or liver impairment, a history of pancreatitis, or who are pregnant or breastfeeding should not begin a ketogenic diet without first speaking to a knowledgeable clinician.
Get a few baseline numbers before you change anything. A fasting blood glucose, fasting insulin, and hemoglobin A1c will show you where your metabolic health stands right now. Pick up a KetoMojo blood ketone and glucose meter for home monitoring.
Fasting insulin. What I look for in patients I'm supporting through cancer: 2 to 3 uIU/mL. That's optimal. Anything above 3 starts pulling away from optimal. Above 6 you're seeing early metabolic dysfunction. Above 10 reflects significant insulin resistance. A lot of women in the 4-to-10 range have been told their labs are normal. Normal is a population average. Optimal is a different number, and in a cancer context that distinction is what informs how I read these markers clinically.
Fasting blood glucose. What I look for: 70 to 85 mg/dL. A reading of 86 to 99 mg/dL falls within the conventional reference range, but in my clinical view it represents early metabolic shift in a direction that warrants attention, particularly in a cancer context. Above 100 mg/dL is, by any clinical definition, metabolic dysfunction. The term "pre-diabetes" is the language conventional medicine uses for this range, and I think it understates what's happening. Cells in this state are glucose-saturated, insulin is chronically elevated, and that internal environment is one cancer cells are well-adapted to. For the full picture of why these numbers shape cancer risk, read What Your Blood Sugar Has to Do With Cancer.
Hemoglobin A1c. What I look for: 5.0% or below. The conventional cutoff of 5.7% is a disease-diagnosis threshold, not a health standard. An A1c of 5.3 or 5.4 reflects chronically elevated average blood glucose that sustains insulin signaling, and in my clinical practice that pattern is what I work to bring down, even when a standard report calls it normal.
Clear your kitchen of the foods that will work against you. Sugar in all its forms, bread, pasta, rice, cereals, crackers, fruit juice, and sweetened products all need to go. Do it practically, as things run out, replace them with something ketogenic.
Download Cronometer to track your food. It is free, accurate, and shows net carbohydrates clearly. Log what you eat for the first two to four weeks. Your target is under 30 grams of net carbs per day.
Plan for the adaptation period. In the first seven to ten days, some people experience headaches, fatigue, irritability, and muscle cramps as the body makes the metabolic shift. This happens almost entirely because of electrolyte loss as the kidneys excrete excess sodium when insulin drops. Drink more water, add salt generously to your food, take magnesium glycinate (200 to 400 mg daily), and eat potassium-rich foods like avocado and leafy greens. These symptoms resolve quickly.
Test your ketones each morning before eating. Write the number down. Over time you will learn exactly which foods and habits keep you in range and which ones pull you out of therapeutic ketosis.
What to eat and what to avoid
Eat freely
Leafy greens: spinach, kale, arugula, Swiss chard
Cruciferous vegetables: broccoli, cauliflower, cabbage
Avocados and avocado oil
Extra virgin olive oil
Pasture-raised eggs
Wild-caught fatty fish: salmon, sardines, mackerel
Grass-fed and grass-finished beef, lamb, and poultry
Full-fat coconut milk and coconut oil
Macadamia nuts, walnuts, pecans
Chia seeds and hemp seeds
Olives and all herbs and spices
Bone broth
Grass-fed butter and ghee
Plain yogurt, kimchi, sauerkraut
Eliminate
Sugar in every form: white, brown, honey, maple syrup, agave
All grains: wheat, rice, oats, corn, quinoa
Bread, pasta, crackers, and cereals
Fruit juice and sports drinks
Most fruit (small portions of berries are acceptable)
Beans and legumes
Root vegetables: potato, sweet potato, parsnip, beet
Processed vegetable oils: canola, soybean, corn oil
Low-fat and fat-free dairy products
Packaged "keto" products with artificial sweeteners
Beer and sweetened alcoholic drinks
A useful daily framework: build every meal around a quality fat, a clean protein, and a generous serving of low-carbohydrate vegetables.
How do your genes affect your response to keto?
Two people can follow the same ketogenic protocol and have noticeably different experiences. A significant part of that comes down to genetics, specifically small variations called single nucleotide polymorphisms, or SNPs (pronounced "snips").
Certain SNPs affect how efficiently you produce ketone bodies, how your body handles saturated versus unsaturated fats, how well you convert plant-based omega-3s into usable forms, and which nutrients you are genetically predisposed to need in higher amounts. The APOE gene influences cardiovascular response to a high-fat diet in a way that is clinically important for a subset of patients. FADS1 and FADS2 variants affect fatty acid conversion in ways that directly shape the best fat choices for your individual body. MTHFR variants affect methylation and detoxification capacity, which becomes clinically significant during cancer treatment.
Knowing your genetic variants removes the guesswork from the protocol. Rather than adjusting based on trial and error, you start with a diet built around your actual biology.
Get your personalized genetic nutrition report
Nutrition Genome offers comprehensive DNA-based analysis that tells you specifically how your genetics interact with diet and nutrients, including how you produce ketones, how you handle dietary fats, and where your individual nutritional needs differ from the average. I use this report clinically with patients to design protocols that work for their actual biology from the start.
Disclosure: I'm a Nutrition Genome affiliate. If you order through this link, I receive a small commission at no additional cost to you. I only recommend testing I use in my own clinical practice.
Recipes to get you started
These six recipes are fully ketogenic, built from anti-inflammatory ingredients, and satisfying enough to stay in regular rotation. Macros are per serving.
Pan-seared salmon with avocado cucumber salad
30 minutes. 2 servings. Anti-inflammatory.
Ingredients:
2 wild-caught salmon fillets (6 oz each)
3 tablespoons extra virgin olive oil
1 lemon, zested and juiced
2 garlic cloves, minced
Fresh dill and flat-leaf parsley
Salt and black pepper
1 large ripe avocado, diced
1 cup cucumber, diced
2 tablespoons red onion, finely chopped
Directions:
Combine 2 tablespoons olive oil, lemon zest, half the lemon juice, garlic, salt, and pepper. Coat the salmon fillets and leave for 10 minutes.
Heat a cast-iron skillet over medium-high heat. Cook salmon 3 to 4 minutes per side until cooked through with a slightly crisped exterior.
Combine avocado, cucumber, and red onion. Dress with remaining olive oil, lemon juice, fresh dill, and a pinch of salt.
Serve salmon over the avocado salad. Scatter flat-leaf parsley over the top.
Per serving: 38g fat, 42g protein, 5g net carbs, about 540 calories.
Baked eggs in creamy garlic spinach
25 minutes. 2 servings. Good on low-appetite days.
Ingredients:
4 large pasture-raised eggs
4 cups fresh spinach
3 tablespoons grass-fed butter or ghee
4 garlic cloves, minced
3/4 cup full-fat coconut cream
1/4 cup grated parmesan
Pinch of nutmeg, salt, and white pepper
Fresh thyme
Directions:
Preheat oven to 375F. Melt butter in an oven-safe skillet over medium heat. Add garlic and stir for one minute.
Add spinach and cook until wilted, about 2 minutes. Pour in coconut cream, nutmeg, salt, and pepper. Stir until gently bubbling.
Create four wells in the spinach and crack an egg into each one. Scatter parmesan over the top.
Transfer to oven and bake 10 to 12 minutes until whites are set and yolks are still slightly runny. Scatter fresh thyme and serve.
Per serving: 41g fat, 22g protein, 6g net carbs, about 480 calories.
Garlic butter chicken thighs with roasted broccoli and lemon tahini
40 minutes. 4 servings. High fat and satisfying.
Ingredients:
4 bone-in, skin-on chicken thighs
4 tablespoons grass-fed butter
4 garlic cloves, minced
1 teaspoon smoked paprika
1 large head broccoli, florets
3 tablespoons extra virgin olive oil
2 tablespoons tahini
Juice of 1 lemon
Salt, pepper, fresh parsley
Directions:
Preheat oven to 425F. Pat chicken dry and season well with salt, pepper, and smoked paprika.
Toss broccoli with 2 tablespoons olive oil and salt. Spread on a baking sheet.
In an oven-safe skillet, melt butter over high heat. Sear chicken skin-side down 5 minutes until golden. Flip, add garlic, cook one more minute.
Transfer skillet and broccoli tray to oven. Roast both 20 to 22 minutes.
Whisk tahini, lemon juice, remaining olive oil, and a splash of water. Drizzle over everything. Finish with fresh parsley.
Per serving: 52g fat, 38g protein, 7g net carbs, about 620 calories.
Cacao cinnamon fat bombs
10 minutes plus chilling. 12 pieces. Anti-inflammatory snack.
Ingredients:
1/2 cup coconut oil, softened
1/4 cup coconut butter
2 tablespoons almond butter, no added sugar
2 tablespoons raw cacao powder
1/2 teaspoon Ceylon cinnamon
1/4 teaspoon vanilla powder or pure extract
Pinch of sea salt
Stevia or monk fruit to taste, optional
Unsweetened shredded coconut or cacao nibs for rolling
Directions:
Stir together all ingredients except the coating until smooth and well combined.
Taste and adjust sweetness with a little stevia or monk fruit if you want it.
Spoon into a silicone mini-muffin tray or roll into small balls by hand.
Roll each piece in shredded coconut or cacao nibs.
Refrigerate at least 30 minutes until firm. Store refrigerated up to two weeks.
Per piece: 14g fat, 1g protein, 1g net carbs, about 135 calories.
Turmeric coconut fat bombs
10 minutes plus chilling. 12 pieces. Anti-inflammatory snack.
Ingredients:
1/2 cup coconut oil, softened
1/4 cup coconut butter
2 tablespoons almond butter, no added sugar
1 teaspoon turmeric powder
1/4 teaspoon black pepper
1/4 teaspoon Ceylon cinnamon
Pinch of ginger powder
Stevia or monk fruit to taste, optional
Unsweetened shredded coconut for rolling
Directions:
Stir together all ingredients except the shredded coconut until smooth and well combined.
Taste and add a small amount of stevia or monk fruit if you want a slightly sweet note.
Spoon into a silicone mini-muffin tray or roll into small balls by hand.
Roll each piece in unsweetened shredded coconut.
Refrigerate at least 30 minutes until firm. Store in the refrigerator for up to two weeks.
Per piece: 14g fat, 1g protein, 1g net carbs, about 135 calories.
Bone broth cauliflower soup with herb oil
35 minutes. 4 servings. Gentle on hard treatment days.
Ingredients:
1 large head cauliflower, roughly chopped
4 cups quality bone broth
4 tablespoons ghee or coconut oil
1 medium onion, diced
4 garlic cloves, chopped
1 cup full-fat coconut milk
1/2 teaspoon turmeric
Salt and white pepper
1/4 cup extra virgin olive oil
Large handful fresh parsley and chives
Directions:
Melt ghee in a large pot over medium heat. Add onion and cook 5 minutes until soft. Add garlic and turmeric, stir one more minute.
Add cauliflower and bone broth. Bring to a boil, reduce heat, and simmer 18 to 20 minutes until very tender.
Add coconut milk. Blend with an immersion blender until completely smooth. Season with salt and white pepper.
Blend olive oil with fresh parsley and chives to make a bright herb oil.
Ladle soup into bowls and finish with a generous swirl of herb oil. Serve warm.
Per serving: 32g fat, 12g protein, 8g net carbs, about 380 calories.
The soup and the baked eggs are the most tolerated when appetite is poor and nausea is present. Have a batch of the fat bombs made in advance so that on days when eating feels impossible, you can still get quality fats and anti-inflammatory compounds into your body throughout the day in very small amounts.
Frequently asked questions
What is the ketogenic diet?
The ketogenic diet is a very low carbohydrate, moderate protein, high fat way of eating that shifts the body from burning glucose to burning fat. Keeping carbohydrates under 30 grams of net carbs per day prompts the liver to produce ketone bodies, putting the body in a metabolic state called ketosis.
How does the ketogenic diet help cancer patients?
Most cancer cells rely almost exclusively on glucose for energy and cannot efficiently use ketone bodies as fuel. This metabolic difference, known as the Warburg Effect, means that lowering blood glucose and raising ketones creates an internal environment that deprives cancer cells of their primary fuel while healthy cells thrive on the alternative energy source.
What are the recommended ketone levels for cancer support?
Nutritional ketosis ranges from 0.5 to 3.0 mmol/L. In my clinical practice, the therapeutic target for cancer support is 2.0 to 3.0 mmol/L, the upper end of nutritional ketosis. This should only be pursued with clinical supervision. Blood glucose targets for people in active treatment are 70 to 85 mg/dL. Testing with a KetoMojo blood ketone meter is the most reliable method.
Can the ketogenic diet reduce chemotherapy side effects?
Research suggests the ketogenic diet may reduce fatigue, improve sleep, lower inflammation, support muscle preservation, and stabilize blood sugar during cancer treatment. A 2025 meta-analysis published in Frontiers in Nutrition found statistically significant improvements in fatigue, insomnia, emotional function, and social function in cancer patients on a ketogenic diet.
Can the ketogenic diet cure cancer?
No. There's no evidence that a ketogenic diet cures cancer on its own, and I don't present it that way. Its role is metabolic support alongside standard treatment, lowering glucose and insulin and raising ketones to target the glucose dependence common to many cancer cells. The clinical research is still developing, and the diet belongs alongside treatment, not in place of it.
Is the ketogenic diet safe during chemotherapy and radiation?
For many people it is, and some evidence suggests it may ease side effects such as fatigue and help stabilize blood sugar during steroid use. Safety depends on your diagnosis, treatment regimen, weight, and nutritional status. People who are underweight or at risk of muscle wasting need a modified approach. It's a decision to make with the clinician overseeing your treatment.
How long does it take to get into ketosis?
Most people reach nutritional ketosis within two to four days of keeping net carbohydrates under 30 grams a day. Becoming fat-adapted, where the body runs efficiently on fat and ketones and energy steadies, usually takes two to four weeks. A blood ketone meter confirms when you're in the 0.5 to 3.0 mmol/L range.
What can you eat on a ketogenic diet for cancer?
Build every meal around a quality fat, a clean protein, and low-carbohydrate vegetables. That means leafy greens and cruciferous vegetables, avocado, extra virgin olive and coconut oil, pasture-raised eggs, wild fatty fish, grass-fed meats, nuts and seeds, and bone broth. Sugar, grains, bread, pasta, most fruit, and starchy vegetables come out.
Who should not follow a ketogenic diet?
People who are underweight or at risk of muscle wasting, and those with Type 1 diabetes, significant kidney or liver impairment, a history of pancreatitis, or who are pregnant or breastfeeding shouldn't start a ketogenic diet without first speaking to a knowledgeable clinician. Some medications, particularly for diabetes, need adjustment before you begin.
Continue reading
Breast Cancer Diet to Reduce Recurrence. Subtype-specific dietary approaches for breast cancer recurrence prevention, including ER+, lobular, TNBC, and HER2+ guidance.
Is Soy Safe After Breast Cancer? The gut microbiome and equol production behind soy's protective effect.
Fasting During Chemotherapy: What the Research Shows. How strategic fasting around treatment protects healthy cells and sensitizes cancer cells.
Work with me
The article is the framework. The application is what I do with patients. I work one-on-one with people navigating cancer who want their care personalized to their actual biology. Your specific diagnosis, your treatment regimen, your medications, your lab values, your genetics, and the life you're trying to live through all of it.
If that's what you're looking for, here's where to start.
About the author
Dr. Lena Suhaila is a naturopathic integrative oncologist and the founder of Naturally Well Within. She holds the FABNO credential, the board certification in naturopathic oncology held by fewer than 200 practitioners worldwide, earned after a CNME-accredited two-year residency with rotations alongside medical, surgical, and radiation oncologists and subsequent work at a major cancer treatment institution now affiliated with City of Hope. Her background includes an undergraduate degree in microbiology. She practices metabolic terrain medicine, functional medicine, and integrative oncology, and she brings Internal Family Systems, polyvagal theory, and Compassionate Inquiry into the work she does with patients. You can work with her directly through the Work With Me page.
References
Warburg O. On the origin of cancer cells. Science. 1956;123(3191):309-314. PMID: 13298683.
Salido-Bueno B, et al. Effects of ketogenic diets on cancer-related variables: a systematic review and meta-analysis of randomised controlled trials. Nutr Bull. 2024;49(3):264-277. PMID: 38923748.
Zhang M, Zhang Q, Huang S, Lu Y, Peng M. Impact of ketogenic diets on cancer patient outcomes: a systematic review and meta-analysis. Front Nutr. 2025;12:1535921. PMID: 40756563.
Klement RJ. Anti-tumor effects of ketogenic diets and their synergism with other treatments in mice: Bayesian evidence synthesis of 1755 individual mouse survival data. Biomed J. 2024;47(1):100609. PMID: 37245566.
Seyfried TN, Flores RE, Poff AM, D'Agostino DP. Cancer as a metabolic disease: implications for novel therapeutics. Carcinogenesis. 2014;35(3):515-527. PMID: 24343361.
Weber DD, et al. Ketogenic diet in the treatment of cancer: where do we stand? Mol Metab. 2020;33:102-121. PMID: 31399389.
Arends J, et al. ESPEN guidelines on nutrition in cancer patients. Clin Nutr. 2017;36(1):11-48. PMID: 27637832.
Cohen CW, et al. A ketogenic diet reduces central obesity and serum insulin in women with ovarian or endometrial cancer. J Nutr. 2018;148(8):1253-1260. PMID: 30137481.
Egashira R, et al. Long-term effects of a ketogenic diet for cancer. Nutrients. 2023;15(10):2334. PMID: 37242217.
Romer M, et al. The use of ketogenic diets in cancer patients: a systematic review. Clin Exp Med. 2021;21(4):501-536. PMID: 33813635.
Amanollahi A, et al. The effect of ketogenic diets on cardio-metabolic outcomes in cancer patients. Nutr Cancer. 2023;75(1):95-111. PMID: 35862121.
Paoli A, et al. Beyond weight loss: a review of the therapeutic uses of very-low-carbohydrate (ketogenic) diets. Eur J Clin Nutr. 2013;67(8):789-796. PMID: 23801097.
